❤️ Digoxin Toxicity: NCLEX Pharmacology Breakdown

Digoxin is a cardiac glycoside used in heart failure and atrial fibrillation — and its toxicity risks make it a favorite on the NCLEX.

It overlaps with:

  • Pharmacology

  • Cardiovascular disorders

  • Electrolyte imbalance

❤️ Digoxin Toxicity: NCLEX Pharmacology Breakdown

📊 Digoxin NCLEX Cheat Sheet

Therapeutic Range 0.5–2.0 ng/mL
Toxic Level Greater than 2.0 ng/mL
Early Signs Nausea, vomiting, anorexia
Visual Symptoms Blurred vision, yellow halos
Cardiac Effects Bradycardia, AV block
Antidote Digoxin Immune Fab (Digibind)
Potassium Role Hypokalemia increases toxicity risk

🧠 NCLEX Tip: Low Potassium = High Digoxin Risk

Hypokalemia increases sensitivity to digoxin, so always check electrolytes — especially in patients on diuretics.

🩺 Nursing Interventions

  • Monitor apical pulse for 1 full minute

  • Hold if HR < 60 bpm (adult), < 100 bpm (infant)

  • Check digoxin blood levels regularly

  • Assess vision and GI symptoms

  • Monitor K⁺ levels, especially with diuretics

  • Keep Digibind on hand for emergencies

💡 Mnemonic: “D.I.G. SLOW”

D Don't give if pulse is low
I Investigate potassium level
G GI signs – nausea, anorexia
S See yellow halos
L Low heart rate = possible toxicity
O Observe for vision changes
W Watch for level >2.0 ng/mL

📲 Learn Cardio Meds with KOTC

  • 🫀 Cardiac pharmacology flashcards

  • 💊 Digoxin vs beta blocker comparisons

  • ⚡ NCLEX-style bradycardia scenarios

Start studying smarter at kingofthecurve.org/studyscience

🏁 Final Thoughts

Digoxin has a narrow therapeutic window and early signs can be subtle.
NCLEX wants you to catch the clues early — before it turns fatal.



 

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